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In the case of Apert syndrome, for example, there is good reason to believe that the causative mutation (in this case, predominantly a 755C-G transversion in the FGFR-2 gene) arises in spermatogonia and is selectively retained in the germinal epithelium because the mutant germ cells enjoy an unspecified selective advantage [4]
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Molecular and functional effects of the novel MEK inhibitor PD0325901 in preclinical models of human leukemias
Phloretin protects bovine rumen epithelial cells from LPS-induced injury
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