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The Core Mechanisms: How GHK-Cu Works at the Cellular Level The most compelling aspect of GHK-Cu is not simply its ability to stimulate collagen, but its broad regulatory effects on gene expression and tissue remodeling pathways

In transsulfuration, the disposal of homocysteine is catalyzed by the B6-dependent cystathionine -synthase, which leads to cystathionine and subsequently to cysteine and GSH biosyntheses (Figure 3)