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Accordingly, we found increased expression of several genes involved with ER-associated degradation, such as Edem1 , Man1a2 and Herpud1 , in mutant growth plates as well as enhanced protein ubiquitination in G6PDH-deficient in vitro differentiated hypoxic chondrocytes (Fig

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The nervous system data is thin compared to the wound healing research, but it keeps this peptide on the radar for neuroscience-adjacent work

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