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For instance, from the perspective of clinical trial research, Samulak et al

GHK-Cu activates various growth factor pathways including transforming growth factor-beta (TGF-) and vascular endothelial growth factor (VEGF), promoting collagen synthesis and angiogenesis

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(14) It is hypothesized that GHK-Cu may influence various biochemical pathways and molecular markers related to inflammation and oxidative stress, although the specific mechanisms remain somewhat uncertain

The clinical relevance is straightforward