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3.7.3 Mechanisms of association between UPF intake and ASD There have been a range of recent studies that have shown that high dietary intake of UPF may contribute to an increased risk of developing ASD, which may be mediated through a range of different pathophysiological mechanisms, including lipid dysregulation, oxidative stress and neuroinflammation (Table 2)

These results established the 2.4mg dose as the primary research target

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