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At the organ level, semaglutide promoted anti-fibrotic and metabolic remodelling by coordinating the downregulation of profibrotic signalling and extracellular matrix protein transcripts, while simultaneously shifting metabolic pathways towards fat oxidation and amino acid catabolism

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Hepatogastroenterology 60 , 10951100 (2013)

Among 16 outcome estimates, 9 reported significant associations