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When Ang II binds to AT1R, it triggers the activation of the JNK and ERK signaling pathways, leading to upregulated expression of early growth response factor 1 (Egr-1), which results in vasoconstriction, inflammation, oxidative stress, and increased expression of ICAM-1 and vascular cell adhesion molecule-1 (VCAM-1)

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The ability of GAL to exert its stimulation of feeding responses may be due to its interactions with other peptide systems